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1m), indicating that KG-induced therapeutic resistance is not simply due to a CCNE1- or MYC-driven replication stress response

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The therapy had a variety of beneficial physiological effects, including the initiation of a rise in glutathione, which might have further contributed to the positive functional effects of the therapy

Different Risk for Hypertension, Diabetes, Dyslipidemia, and Hyperuricemia According to Level of Body Mass Index in Japanese and American Subjects