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In diesen Modellen berichteten Forscher von einer schnelleren Organisation des Reparaturgewebes, einer verbesserten strukturellen Erholung sowie einer gesteigerten Fibroblastenaktivitt bei verletzten Tieren, was zusammen die Grundlage des breit untersuchten regenerativen Profils der Substanz bildet

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also known as MAP3K7), p38 mitogen-activated protein kinase (p38 MAPK), RHO (RAS homolog)-like GTPase signaling pathways, phosphoinositide 3-kinase (PI3K)AKT (also known as protein kinase B), extracellular signal-regulated kinase (ERK), JUN N-terminal kinase (JNK) or nuclear factor-B (NF-B)

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